Max in WT synaptoneurosomes, suggesting that Src signaling can be downregulated in KI synapses. Alternatively, our power to rescue SERT operate in KI midbrain synaptoneurosomes with the inhibition of FAK implies elevated FAK signaling downstream of the Pro32Pro33 mutant, as confirmed by elevated pFAK localization in 5-HT synapses. Our knowledge https://leonardob333cvr7.wikiexcerpt.com/user